Identification of Fusion Gene Breakpoints is Feasible and Facilitates Accurate Sensitive Minimal Residual Disease Monitoring on Genomic Level in Patients With PML-RARA, CBFB-MYH11, and RUNX1-RUNX1T1

Lignende værker

• Genomic landscape of pediatric B-other acute lymphoblastic leukemia in a consecutive European cohort
  af: Zaliova, Marketa, et al.
  Udgivet: (2019)
• AML associated oncofusion proteins PML-RARA, AML1-ETO and CBFB-MYH11 target RUNX/ETS-factor binding sites to modulate H3ac levels and drive leukemogenesis
  af: Singh, Abhishek A., et al.
  Udgivet: (2016)
• Runx1 is required for hematopoietic defects and leukemogenesis in Cbfb-MYH11 knockin mice
  af: Hyde, R. Katherine, et al.
  Udgivet: (2015)
• Frequency and prognostic impact of ZEB2 H1038 and Q1072 mutations in childhood B-other acute lymphoblastic leukemia
  af: Zaliova, Marketa, et al.
  Udgivet: (2020)
• RUNX1 Repression Independent Mechanisms of Leukemogenesis by Fusion Genes CBFB-MYH11 and AML1-ETO (RUNX1-RUNX1T1)
  af: Hyde, R. Katherine, et al.
  Udgivet: (2010)