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Suppression of non-homologous end joining does not rescue DNA repair defects in Fanconi anemia patient cells

Severe cellular sensitivity and aberrant chromosomal rearrangements in response to DNA interstrand crosslink (ICL) inducing agents are hallmarks of Fanconi anemia (FA) deficient cells. These phenotypes have previously been ascribed to inappropriate activity of non-homologous end joining (NHEJ) rathe...

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Bibliografiske detaljer
Udgivet i:Cell Cycle
Main Authors: Thongthip, Supawat, Conti, Brooke A., Lach, Francis P., Smogorzewska, Agata
Format: Artigo
Sprog:Inglês
Udgivet: Taylor & Francis 2020
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Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7553586/
https://ncbi.nlm.nih.gov/pubmed/32865112
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2020.1810394
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