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Suppression of non-homologous end joining does not rescue DNA repair defects in Fanconi anemia patient cells

Severe cellular sensitivity and aberrant chromosomal rearrangements in response to DNA interstrand crosslink (ICL) inducing agents are hallmarks of Fanconi anemia (FA) deficient cells. These phenotypes have previously been ascribed to inappropriate activity of non-homologous end joining (NHEJ) rathe...

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Publicat a:Cell Cycle
Autors principals: Thongthip, Supawat, Conti, Brooke A., Lach, Francis P., Smogorzewska, Agata
Format: Artigo
Idioma:Inglês
Publicat: Taylor & Francis 2020
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7553586/
https://ncbi.nlm.nih.gov/pubmed/32865112
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2020.1810394
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