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Suppression of non-homologous end joining does not rescue DNA repair defects in Fanconi anemia patient cells
Severe cellular sensitivity and aberrant chromosomal rearrangements in response to DNA interstrand crosslink (ICL) inducing agents are hallmarks of Fanconi anemia (FA) deficient cells. These phenotypes have previously been ascribed to inappropriate activity of non-homologous end joining (NHEJ) rathe...
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| Publicat a: | Cell Cycle |
|---|---|
| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Taylor & Francis
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7553586/ https://ncbi.nlm.nih.gov/pubmed/32865112 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2020.1810394 |
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