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Transient activation of mTORC1 signaling in skeletal muscle is independent of Akt1 regulation
The regulation of cellular protein synthesis is a critical determinant of skeletal muscle growth and hypertrophy in response to an increased workload such as resistance exercise. The mechanistic target of rapamycin complex 1 (mTORC1) and its upstream protein kinase Akt1 have been implicated as a cen...
שמור ב:
| הוצא לאור ב: | Physiol Rep |
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| Main Authors: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
John Wiley and Sons Inc.
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7547586/ https://ncbi.nlm.nih.gov/pubmed/33038070 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14599 |
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