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Transient activation of mTORC1 signaling in skeletal muscle is independent of Akt1 regulation
The regulation of cellular protein synthesis is a critical determinant of skeletal muscle growth and hypertrophy in response to an increased workload such as resistance exercise. The mechanistic target of rapamycin complex 1 (mTORC1) and its upstream protein kinase Akt1 have been implicated as a cen...
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| Publicat a: | Physiol Rep |
|---|---|
| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
John Wiley and Sons Inc.
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7547586/ https://ncbi.nlm.nih.gov/pubmed/33038070 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14599 |
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