Correction: Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity

Podobne zapisy

• Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity
  od: Watson, Adrienne L., i wsp.
  Wydane: (2014)
• PTEN and NF1 inactivation in Schwann cells produces a severe phenotype in the peripheral nervous system that promotes the development and malignant progression of peripheral nerve sheath tumors
  od: Keng, Vincent W., i wsp.
  Wydane: (2012)
• Canonical Wnt/β-catenin Signaling Drives Human Schwann Cell Transformation, Progression, and Tumor Maintenance
  od: Watson, Adrienne L., i wsp.
  Wydane: (2013)
• Conditional Inactivation of Pten with EGFR Overexpression in Schwann Cells Models Sporadic MPNST
  od: Keng, Vincent W., i wsp.
  Wydane: (2012)
• Interweaving the strands: β-Catenin, an HIV Co-Receptor, and Schwann Cell Tumors
  od: Largaespada, David, i wsp.
  Wydane: (2013)