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Mutational resilience of antiviral restriction favors primate TRIM5α in host-virus evolutionary arms races

Host antiviral proteins engage in evolutionary arms races with viruses, in which both sides rapidly evolve at interaction interfaces to gain or evade immune defense. For example, primate TRIM5α uses its rapidly evolving ‘v1’ loop to bind retroviral capsids, and single mutations in this loop can dram...

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Bibliografski detalji
Izdano u:eLife
Glavni autori: Tenthorey, Jeannette L, Young, Candice, Sodeinde, Afeez, Emerman, Michael, Malik, Harmit S
Format: Artigo
Jezik:Inglês
Izdano: eLife Sciences Publications, Ltd 2020
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC7492085/
https://ncbi.nlm.nih.gov/pubmed/32930662
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.59988
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