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A mechanistic hypothesis for the impairment of synaptic plasticity by soluble Aβ oligomers from Alzheimer brain
It is increasingly accepted that early cognitive impairment in Alzheimer’s disease results in considerable part from synaptic dysfunction caused by the accumulation of a range of oligomeric assemblies of amyloid β-protein (Aβ). Most studies have used synthetic Aβ peptides to explore the mechanisms o...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Neurochem |
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| Κύριοι συγγραφείς: | , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2020
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7487043/ https://ncbi.nlm.nih.gov/pubmed/32180217 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jnc.15007 |
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