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Soluble Oligomers of the Amyloid β-Protein Impair Synaptic Plasticity and Behavior

During the last 25 years, neuropathological, biochemical, genetic, cell biological and even therapeutic studies in humans have all supported the hypothesis that the gradual cerebral accumulation of soluble and insoluble assemblies of the amyloid β-protein (Aβ) in limbic and association cortices trig...

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Detalles Bibliográficos
Autor Principal: Selkoe, Dennis J.
Formato: Artigo
Idioma:Inglês
Publicado: 2008
Assuntos:
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2601528/
https://ncbi.nlm.nih.gov/pubmed/18359102
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbr.2008.02.016
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