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Soluble Oligomers of the Amyloid β-Protein Impair Synaptic Plasticity and Behavior
During the last 25 years, neuropathological, biochemical, genetic, cell biological and even therapeutic studies in humans have all supported the hypothesis that the gradual cerebral accumulation of soluble and insoluble assemblies of the amyloid β-protein (Aβ) in limbic and association cortices trig...
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2601528/ https://ncbi.nlm.nih.gov/pubmed/18359102 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbr.2008.02.016 |
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