Pressure overload generates a cardiac-specific profile of inflammatory mediators

Mechanisms that contribute to myocardial fibrosis, particularly in response to left ventricular pressure overload (LVPO), remain poorly defined. To test the hypothesis that a myocardial-specific profile of secreted factors is produced in response to PO, levels of 44 factors implicated in immune cell...

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Detalhes bibliográficos
Publicado no:Am J Physiol Heart Circ Physiol
Main Authors: O’Brien, Matthew, Baicu, Catalin F., Van Laer, An O., Zhang, Yuhua, McDonald, Lindsey T., LaRue, Amanda C., Zile, Michael R., Bradshaw, Amy D.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2020
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7473927/
https://ncbi.nlm.nih.gov/pubmed/32589444
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00274.2020
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