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Simultaneous targeting of mitochondria and monocytes enhances neuroprotection against ischemia–reperfusion injury
Ischemia–reperfusion injury impairs the efficacy of reperfusion therapy after ischemic stroke. Cyclophilin D (CypD)-mediated openings of mitochondrial permeability transition pore (mPTP) and subsequent monocyte-mediated inflammation are considered as major mechanisms of reperfusion injury. However,...
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| 出版年: | Sci Rep |
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| 主要な著者: | , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group UK
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7468234/ https://ncbi.nlm.nih.gov/pubmed/32879367 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-71326-x |
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