iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin

BACKGROUND: Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (mdx mice) and that both neuronal NOS (nNOS) and inducible NOS (iNOS) might contribute to the reaction because nNOS is mislocalized in the...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:BMC Musculoskelet Disord
Päätekijät: Nogami, Ken’ichiro, Maruyama, Yusuke, Elhussieny, Ahmed, Sakai-Takemura, Fusako, Tanihata, Jun, Kira, Jun-ichi, Miyagoe-Suzuki, Yuko, Takeda, Shin’ichi
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: BioMed Central 2020
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7374827/
https://ncbi.nlm.nih.gov/pubmed/32693782
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12891-020-03501-0
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