טוען...
iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin
BACKGROUND: Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (mdx mice) and that both neuronal NOS (nNOS) and inducible NOS (iNOS) might contribute to the reaction because nNOS is mislocalized in the...
שמור ב:
| הוצא לאור ב: | BMC Musculoskelet Disord |
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| Main Authors: | , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
BioMed Central
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7374827/ https://ncbi.nlm.nih.gov/pubmed/32693782 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12891-020-03501-0 |
| תגים: |
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