Site-specific acetyl-mimetic modification of cardiac troponin I modulates myofilament relaxation and calcium sensitivity

OBJECTIVE: Cardiac troponin I (cTnI) is an essential physiological and pathological regulator of cardiac relaxation. Significant to this regulation, the post-translational modification of cTnI through phosphorylation functions as a key mechanism to accelerate myofibril relaxation. Similar to phospho...

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Publié dans:J Mol Cell Cardiol
Auteurs principaux: Lin, Ying H., Schmidt, William, Fritz, Kristofer S., Jeong, Mark Y., Cammarato, Anthony, Foster, D. Brian, Biesiadecki, Brandon J., McKinsey, Timothy A., Woulfe, Kathleen C.
Format: Artigo
Langue:Inglês
Publié: 2020
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Article
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC7363438/
https://ncbi.nlm.nih.gov/pubmed/31981571
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2020.01.007
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