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Fibrillization of 40-residue β-Amyloid Peptides in Membrane-Like Environments Leads to Different Fibril Structures and Reduced Molecular Polymorphisms

The molecular-level polymorphism in β-Amyloid (Aβ) fibrils have recently been considered as a pathologically relevant factor in Alzheimer’s disease (AD). Studies showed that the structural deviations in human-brain-seeded Aβ fibrils potentially correlated with the clinical histories of AD patients....

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Publicat a:Biomolecules
Autors principals: Cheng, Qinghui, Hu, Zhi-Wen, Tobin-Miyaji, Yuto, Perkins, Amy E., Deak, Terrence, Qiang, Wei
Format: Artigo
Idioma:Inglês
Publicat: MDPI 2020
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7356566/
https://ncbi.nlm.nih.gov/pubmed/32521743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/biom10060881
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