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Intracellular acidification facilitates receptor-operated TRPC4 activation through PLCδ1 in a Ca(2+)-dependent manner
Transient Receptor Potential Canonical 4 (TRPC4) forms non-selective cation channels activated downstream from receptors that signal through G proteins. Our recent work suggests that TRPC4 channels are particularly coupled to pertussis toxin-sensitive G(i/o) proteins, with a co-dependence on phospho...
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| Publicat a: | J Physiol |
|---|---|
| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7332378/ https://ncbi.nlm.nih.gov/pubmed/32338378 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP279658 |
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