IL-17 Induced Autophagy Regulates Mitochondrial Dysfunction and Fibrosis in Severe Asthmatic Bronchial Fibroblasts

The accumulation of fibroblasts, their synthesis of extracellular matrix (ECM) proteins and their innate resistance to apoptosis are characteristics of subepithelial fibrosis observed in severe asthma. Interleukin-17 (IL-17) is an important regulator of airway remodeling in asthma. However, the cont...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Front Immunol
Päätekijät: Ramakrishnan, Rakhee K., Bajbouj, Khuloud, Al Heialy, Saba, Mahboub, Bassam, Ansari, Abdul Wahid, Hachim, Ibrahim Y., Rawat, Surendra, Salameh, Laila, Hachim, Mahmood Y., Olivenstein, Ronald, Halwani, Rabih, Hamoudi, Rifat, Hamid, Qutayba
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Frontiers Media S.A. 2020
Aiheet:
Immunology
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7326148/
https://ncbi.nlm.nih.gov/pubmed/32670268
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2020.01002
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