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Carboxy-terminal truncation and phosphorylation of α-synuclein elongates survival in a prion-like seeding mouse model of synucleinopathy
Pathologic intracellular inclusions formed from polymers of misfolded α-synuclein (αsyn) protein define a group of neurodegenerative diseases termed synucleinopathies which includes Parkinson’s disease (PD). Prion-like recruitment of endogenous cellular αsyn has been demonstrated to occur in animal...
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| Pubblicato in: | Neurosci Lett |
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| Autori principali: | , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2020
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7323740/ https://ncbi.nlm.nih.gov/pubmed/32371157 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neulet.2020.135017 |
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