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Carboxy-terminal truncations of mouse alpha-synuclein alter aggregation and prion-like seeding
α-synuclein (αsyn) forms pathologic inclusions in several neurodegenerative diseases termed synucleinopathies. The inclusions are comprised of αsyn fibrils harboring prion-like properties. Prion-like activity of αsyn has been studied by intra-cerebral injection of fibrils into mice, where the presen...
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| Vydáno v: | FEBS Lett |
|---|---|
| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2020
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7188589/ https://ncbi.nlm.nih.gov/pubmed/31912891 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/1873-3468.13728 |
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