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Carboxy-terminal truncations of mouse alpha-synuclein alter aggregation and prion-like seeding
α-synuclein (αsyn) forms pathologic inclusions in several neurodegenerative diseases termed synucleinopathies. The inclusions are comprised of αsyn fibrils harboring prion-like properties. Prion-like activity of αsyn has been studied by intra-cerebral injection of fibrils into mice, where the presen...
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| Publicado no: | FEBS Lett |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7188589/ https://ncbi.nlm.nih.gov/pubmed/31912891 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/1873-3468.13728 |
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