APOL1 Kidney-Risk Variants Induce Mitochondrial Fission

INTRODUCTION: APOL1 G1 and G2 nephropathy-risk variants cause mitochondrial dysfunction and contribute to kidney disease. Analyses were performed to determine the genetic regulation of APOL1 and elucidate potential mechanisms in APOL1-nephropathy. METHODS: A global gene expression analysis was perfo...

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Detalhes bibliográficos
Publicado no:Kidney Int Rep
Main Authors: Ma, Lijun, Ainsworth, Hannah C., Snipes, James A., Murea, Mariana, Choi, Young A, Langefeld, Carl D., Parks, John S., Bharadwaj, Manish S., Chou, Jeff W., Hemal, Ashok K., Petrovic, Snezana, Craddock, Ann L., Cheng, Dongmei, Hawkins, Gregory A., Miller, Lance D., Hicks, Pamela J., Saleem, Moin A., Divers, Jasmin, Molina, Anthony J.A., Freedman, Barry I.
Formato: Artigo
Idioma:Inglês
Publicado em: Elsevier 2020
Assuntos:
Translational Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7271005/
https://ncbi.nlm.nih.gov/pubmed/32518871
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ekir.2020.03.020
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