Loss of FOXM1 in macrophages promotes pulmonary fibrosis by activating p38 MAPK signaling pathway

Idiopathic pulmonary fibrosis (IPF) is a chronic disease with high mortality and is refractory to treatment. Pulmonary macrophages can both promote and repress fibrosis, however molecular mechanisms regulating macrophage functions during fibrosis remain poorly understood. FOXM1 is a transcription fa...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:PLoS Genet
Päätekijät: Goda, Chinmayee, Balli, David, Black, Markaisa, Milewski, David, Le, Tien, Ustiyan, Vladimir, Ren, Xiaomeng, Kalinichenko, Vladimir V., Kalin, Tanya V.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2020
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7173935/
https://ncbi.nlm.nih.gov/pubmed/32271749
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1008692
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