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Targeting BRD/BET proteins inhibits adaptive kinome upregulation and enhances the effects of BRAF/MEK inhibitors in melanoma

BACKGROUND: BRAF-mutant melanoma patients respond to BRAF inhibitors and MEK inhibitors (BRAFi/MEKi), but drug-tolerant cells persist, which may seed disease progression. Adaptive activation of receptor tyrosine kinases (RTKs) has been associated with melanoma cell drug tolerance following targeted...

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Publicat a:Br J Cancer
Autors principals: Tiago, Manoela, Capparelli, Claudia, Erkes, Dan A., Purwin, Timothy J., Heilman, Shea A., Berger, Adam C., Davies, Michael A., Aplin, Andrew E.
Format: Artigo
Idioma:Inglês
Publicat: Nature Publishing Group UK 2020
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7078299/
https://ncbi.nlm.nih.gov/pubmed/31932756
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41416-019-0724-y
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