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Smad4 promotes diabetic nephropathy by modulating glycolysis and OXPHOS
Diabetic nephropathy (DN) is the leading cause of end‐stage kidney disease. TGF‐β1/Smad3 signalling plays a major pathological role in DN; however, the contribution of Smad4 has not been examined. Smad4 depletion in the kidney using anti‐Smad4 locked nucleic acid halted progressive podocyte damage a...
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| Vydáno v: | EMBO Rep |
|---|---|
| Hlavní autoři: | , , , , , , , , , , , , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2020
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7001498/ https://ncbi.nlm.nih.gov/pubmed/31916354 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embr.201948781 |
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