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Hypertension-causing cullin 3 mutations disrupt COP9 signalosome binding
The discovery of new genetic mutations that cause hypertension has illuminated previously unrecognized physiological pathways. One such regulatory pathway was identified when mutations in with no lysine kinase (WNK)4, Kelch-like 3 (KLHL3), and cullin 3 (CUL3) were shown to cause the disease familial...
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| Udgivet i: | Am J Physiol Renal Physiol |
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| Main Authors: | , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
American Physiological Society
2020
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6985821/ https://ncbi.nlm.nih.gov/pubmed/31813255 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00497.2019 |
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