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Regulation of NKCC2B by TNF-α in response to salt restriction
We have previously shown that TNF-α produced by renal epithelial cells inhibits Na(+)-K(+)-2Cl(−) cotransporter (NKCC2) activity as part of a mechanism that attenuates increases in blood pressure in response to high NaCl intake. As the role of TNF-α in the kidney is still being defined, the effects...
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| 出版年: | Am J Physiol Renal Physiol |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6985818/ https://ncbi.nlm.nih.gov/pubmed/31813248 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00388.2019 |
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