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Renal-specific silencing of TNF unmasks salt-dependent increases in blood pressure via an NKCC2A-dependent mechanism

We tested the hypothesis that tumor necrosis factor-alpha (TNF) produced within the kidney and acting on the renal tubular system is part of a regulatory mechanism that attenuates increases in blood pressure in response to high salt intake. Intrarenal administration of a lentivirus construct, which...

詳細記述

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書誌詳細
出版年:Hypertension
主要な著者: Hao, Shoujin, Hao, Mary, Ferreri, Nicholas R.
フォーマット: Artigo
言語:Inglês
出版事項: 2018
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC6027635/
https://ncbi.nlm.nih.gov/pubmed/29735631
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.117.10764
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