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Renal-specific silencing of TNF unmasks salt-dependent increases in blood pressure via an NKCC2A-dependent mechanism
We tested the hypothesis that tumor necrosis factor-alpha (TNF) produced within the kidney and acting on the renal tubular system is part of a regulatory mechanism that attenuates increases in blood pressure in response to high salt intake. Intrarenal administration of a lentivirus construct, which...
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| 出版年: | Hypertension |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6027635/ https://ncbi.nlm.nih.gov/pubmed/29735631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.117.10764 |
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