Loss of endothelial sulfatase-1 after experimental sepsis attenuates subsequent pulmonary inflammatory responses

Sepsis patients are at increased risk for hospital-acquired pulmonary infections, potentially due to postseptic immunosuppression known as the compensatory anti-inflammatory response syndrome (CARS). CARS has been attributed to leukocyte dysfunction, with an unclear role for endothelial cells. The p...

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Detalhes bibliográficos
Publicado no:Am J Physiol Lung Cell Mol Physiol
Main Authors: Oshima, Kaori, Han, Xiaorui, Ouyang, Yilan, El Masri, Rana, Yang, Yimu, Haeger, Sarah M., McMurtry, Sarah A., Lane, Trevor C., Davizon-Castillo, Pavel, Zhang, Fuming, Yue, Xinping, Vivès, Romain R., Linhardt, Robert J., Schmidt, Eric P.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2019
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6879902/
https://ncbi.nlm.nih.gov/pubmed/31461325
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00175.2019
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