Bax Regulates Neuronal Ca(2+) Homeostasis

Excessive Ca(2+) entry during glutamate receptor overactivation (“excitotoxicity”) induces acute or delayed neuronal death. We report here that deficiency in bax exerted broad neuroprotection against excitotoxic injury and oxygen/glucose deprivation in mouse neocortical neuron cultures and reduced i...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: D'Orsi, Beatrice, Kilbride, Seán M., Chen, Gang, Perez Alvarez, Sergio, Bonner, Helena P., Pfeiffer, Shona, Plesnila, Nikolaus, Engel, Tobias, Henshall, David C., Düssmann, Heiko, Prehn, Jochen H.M.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2015
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6795257/
https://ncbi.nlm.nih.gov/pubmed/25632145
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2453-14.2015
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