Crosstalk between the Akt/mTORC1 and NF-κB signaling pathways promotes hypoxia-induced pulmonary hypertension by increasing DPP4 expression in PASMCs

Abnormal wound healing by pulmonary artery smooth muscle cells (PASMCs) promotes vascular remodeling in hypoxia-induced pulmonary hypertension (HPH). Increasing evidence shows that both the mammalian target of rapamycin complex 1 (mTORC1) and nuclear factor-kappa B (NF-κB) are involved in the develo...

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Publicado no:Acta Pharmacol Sin
Main Authors: Li, Ying, Yang, Li, Dong, Liang, Yang, Zhi-wei, Zhang, Jing, Zhang, Sheng-li, Niu, Meng-jie, Xia, Jing-wen, Gong, Yi, Zhu, Ning, Zhang, Xiu-juan, Zhang, Yuan-yuan, Wei, Xiao-min, Zhang, You-zhi, Zhang, Peng, Li, Sheng-qing
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6786428/
https://ncbi.nlm.nih.gov/pubmed/31316183
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41401-019-0272-2
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