ロード中...
Crosstalk between the Akt/mTORC1 and NF-κB signaling pathways promotes hypoxia-induced pulmonary hypertension by increasing DPP4 expression in PASMCs
Abnormal wound healing by pulmonary artery smooth muscle cells (PASMCs) promotes vascular remodeling in hypoxia-induced pulmonary hypertension (HPH). Increasing evidence shows that both the mammalian target of rapamycin complex 1 (mTORC1) and nuclear factor-kappa B (NF-κB) are involved in the develo...
保存先:
| 出版年: | Acta Pharmacol Sin |
|---|---|
| 主要な著者: | , , , , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group UK
2019
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6786428/ https://ncbi.nlm.nih.gov/pubmed/31316183 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41401-019-0272-2 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|