α-1-Antichymotrypsin Promotes β-Sheet Amyloid Plaque Deposition in a Transgenic Mouse Model of Alzheimer's Disease

Documenti analoghi

• Microglial Activation and β-Amyloid Deposit Reduction Caused by a Nitric Oxide-Releasing Nonsteroidal Anti-Inflammatory Drug in Amyloid Precursor Protein Plus Presenilin-1 Transgenic Mice
  di: Jantzen, Paul T., et al.
  Pubblicazione: (2002)
• Intracranial administration of deglycosylated C-terminal-specific anti-Aβ antibody efficiently clears amyloid plaques without activating microglia in amyloid-depositing transgenic mice
  di: Carty, Niki C, et al.
  Pubblicazione: (2006)
• Passive Amyloid Immunotherapy Clears Amyloid and Transiently Activates Microglia in a Transgenic Mouse Model of Amyloid Deposition
  di: Wilcock, Donna M., et al.
  Pubblicazione: (2004)
• Intracranially Administered Anti-Αβ Antibodies Reduce β-Amyloid Deposition by Mechanisms Both Independent of and Associated with Microglial Activation
  di: Wilcock, Donna M., et al.
  Pubblicazione: (2003)
• Amyloid-β protofibril levels correlate with spatial learning in Arctic Alzheimer’s disease transgenic mice
  di: Lord, Anna, et al.
  Pubblicazione: (2009)