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Compromised Hemodynamic Response in Amyloid Precursor Protein Transgenic Mice

APP23 transgenic mice overexpressing amyloid precursor protein (APP(751)) reproduce neuropathological changes associated with Alzheimer's disease such as high levels of amyloid plaques, cerebral amyloid angiopathy, and associated vascular pathologies. Functional magnetic resonance imaging (fMRI...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:J Neurosci
Päätekijät: Mueggler, Thomas, Sturchler-Pierrat, Christine, Baumann, Diana, Rausch, Martin, Staufenbiel, Matthias, Rudin, Markus
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2002
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6757895/
https://ncbi.nlm.nih.gov/pubmed/12177216
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-16-07218.2002
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