GSE4 peptide suppresses oxidative and telomere deficiencies in ataxia telangiectasia patient cells

Ataxia telangiectasia (AT) is a genetic disease caused by mutations in the ATM gene but the mechanisms underlying AT are not completely understood. Key functions of the ATM protein are to sense and regulate cellular redox status and to transduce DNA double-strand break signals to downstream effector...

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Udgivet i:Cell Death Differ
Main Authors: Pintado-Berninches, Laura, Fernandez-Varas, Beatriz, Benitez-Buelga, Carlos, Manguan-Garcia, Cristina, Serrano-Benitez, Almudena, Iarriccio, Laura, Carrillo, Jaime, Guenechea, Guillermo, Egusquiaguirre, Susana P., Pedraz, Jose-Luis, Hernández, Rosa M., Igartua, Manoli, Arias-Salgado, Elena G., Cortés-Ledesma, Felipe, Sastre, Leandro, Perona, Rosario
Format: Artigo
Sprog:Inglês
Udgivet: Nature Publishing Group UK 2019
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Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6748109/
https://ncbi.nlm.nih.gov/pubmed/30670828
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41418-018-0272-7
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