miR-146a attenuates apoptosis and modulates autophagy by targeting TAF9b/P53 pathway in doxorubicin-induced cardiotoxicity

Clinical therapy of doxorubicin (DOX) is limited due to its cardiotoxicity. miR-146a was proved as a protective factor in many cardiovascular diseases, but its role in chronic DOX-induced cardiotoxicity is unclear. The objective of this study was to demonstrate the role of miR-146a in low-dose long-...

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Detaylı Bibliyografya
Yayımlandı:Cell Death Dis
Asıl Yazarlar: Pan, Jian-An, Tang, Yong, Yu, Jian-Ying, Zhang, Hui, Zhang, Jun-Feng, Wang, Chang-Qian, Gu, Jun
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Nature Publishing Group UK 2019
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6739392/
https://ncbi.nlm.nih.gov/pubmed/31511497
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1901-x
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