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Activation of Nrf2 by miR-152 Inhibits Doxorubicin-Induced Cardiotoxicity via Attenuation of Oxidative Stress, Inflammation, and Apoptosis

Doxorubicin (DOX) could trigger congestive heart failure, which largely limited the clinical use of DOX. microRNAs (miRNAs) were closely involved in the pathogenesis of DOX-induced cardiomyopathy. Here, we aimed to investigate the effect of miR-152 on DOX-induced cardiotoxicity in mice. To study thi...

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Vydáno v:Oxid Med Cell Longev
Hlavní autoři: Zhang, Wen-Bin, Lai, Xin, Guo, Xu-Feng
Médium: Artigo
Jazyk:Inglês
Vydáno: Hindawi 2021
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC7857911/
https://ncbi.nlm.nih.gov/pubmed/33574984
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2021/8860883
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