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Mitochondrial translocation of cyclin C stimulates intrinsic apoptosis through Bax recruitment
Intrinsic apoptosis requires mitochondrial outer membrane disruption triggered by recruitment, activation, and oligomerization of the Bcl‐2 homology protein Bax. Following oxidative stress, we demonstrated that the transcriptional regulator cyclin C is released into the cytosol where it directs mito...
Uloženo v:
| Vydáno v: | EMBO Rep |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6726902/ https://ncbi.nlm.nih.gov/pubmed/31385392 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embr.201847425 |
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