Zn(2+) Inhibits Mitochondrial Movement in Neurons by Phosphatidylinositol 3-Kinase Activation

Mitochondria have been identified as targets of the neurotoxic actions of zinc, possibly through decreased mitochondrial energy production and increased reactive oxygen species accumulation. It has been hypothesized that impairment of mitochondrial trafficking may be a mechanism of neuronal injury....

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Vydáno v:J Neurosci
Hlavní autoři: Malaiyandi, Latha M., Honick, Anthony S., Rintoul, Gordon L., Wang, Qiming J., Reynolds, Ian J.
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 2005
Témata:
Cellular/Molecular
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6725691/
https://ncbi.nlm.nih.gov/pubmed/16221861
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0868-05.2005
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