ARF6 and AMAP1 are major targets of KRAS and TP53 mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer

Although KRAS and TP53 mutations are major drivers of pancreatic ductal adenocarcinoma (PDAC), the incurable nature of this cancer still remains largely elusive. ARF6 and its effector AMAP1 are often overexpressed in different cancers and regulate the intracellular dynamics of integrins and E-cadher...

詳細記述

保存先:
書誌詳細
出版年:Proc Natl Acad Sci U S A
主要な著者: Hashimoto, Shigeru, Furukawa, Shotaro, Hashimoto, Ari, Tsutaho, Akio, Fukao, Akira, Sakamura, Yurika, Parajuli, Gyanu, Onodera, Yasuhito, Otsuka, Yutaro, Handa, Haruka, Oikawa, Tsukasa, Hata, Soichiro, Nishikawa, Yoshihiro, Mizukami, Yusuke, Kodama, Yuzo, Murakami, Masaaki, Fujiwara, Toshinobu, Hirano, Satoshi, Sabe, Hisataka
フォーマット: Artigo
言語:Inglês
出版事項: National Academy of Sciences 2019
主題:
PNAS Plus
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC6717289/
https://ncbi.nlm.nih.gov/pubmed/31399545
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1901765116
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