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The Ca(2+)-Activated K(+) Channel KCNN4/KCa3.1 Contributes to Microglia Activation and Nitric Oxide-Dependent Neurodegeneration
Brain damage and disease involve activation of microglia and production of potentially neurotoxic molecules, but there are no treatments that effectively target their harmful properties. We present evidence that the small-conductance Ca(2+)/calmodulin-activated K(+) channel KCNN4/ KCa3.1/SK4/IK1 is...
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| Publicado no: | J Neurosci |
|---|---|
| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Society for Neuroscience
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6672279/ https://ncbi.nlm.nih.gov/pubmed/17202491 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3593-06.2007 |
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