Inhibition of the Ca(2+)-Dependent K(+) Channel, KCNN4/KCa3.1, Improves Tissue Protection and Locomotor Recovery after Spinal Cord Injury

Spinal cord injury (SCI) triggers inflammatory responses that involve neutrophils, macrophages/microglia and astrocytes and molecules that potentially cause secondary tissue damage and functional impairment. Here, we assessed the contribution of the calcium-dependent K(+) channel KCNN4 (KCa3.1, IK1,...

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Vydáno v:J Neurosci
Hlavní autoři: Bouhy, Delphine, Ghasemlou, Nader, Lively, Starlee, Redensek, Adriana, Rathore, Khizr I., Schlichter, Lyanne C., David, Samuel
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 2011
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6633238/
https://ncbi.nlm.nih.gov/pubmed/22072681
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0047-11.2011
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