Amyloid β Oligomers (Aβ(1–42) Globulomer) Suppress Spontaneous Synaptic Activity by Inhibition of P/Q-Type Calcium Currents

Abnormal accumulation of soluble oligomers of amyloid β (Aβ) is believed to cause malfunctioning of neurons in Alzheimer's disease. It has been shown that Aβ oligomers impair synaptic plasticity, thereby altering the ability of the neuron to store information. We examined the underlying cellula...

Ful tanımlama

Kaydedildi:
Detaylı Bibliyografya
Yayımlandı:J Neurosci
Asıl Yazarlar: Nimmrich, Volker, Grimm, Christiane, Draguhn, Andreas, Barghorn, Stefan, Lehmann, Alexander, Schoemaker, Hans, Hillen, Heinz, Gross, Gerhard, Ebert, Ulrich, Bruehl, Claus
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2008
Konular:
Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6671006/
https://ncbi.nlm.nih.gov/pubmed/18216187
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4771-07.2008
Etiketler: Etiketle
Etiket eklenmemiş, İlk siz ekleyin!

Benzer Materyaller