The Beta Amyloid Dysfunction (BAD) Hypothesis for Alzheimer’s Disease

Por Hillen, Heinz
Publicado no Front Neurosci (2019)

Immunotherapy alleviates amyloid-associated synaptic pathology in an Alzheimer’s disease mouse model

Por Dorostkar, Mario M., Burgold, Steffen, Filser, Severin, Barghorn, Stefan, Schmidt, Boris, Anumala, Upendra Rao, Hillen, Heinz, Klein, Corinna, Herms, Jochen
Publicado no Brain (2014)

Amyloid β Oligomers (Aβ(1–42) Globulomer) Suppress Spontaneous Synaptic Activity by Inhibition of P/Q-Type Calcium Currents

Por Nimmrich, Volker, Grimm, Christiane, Draguhn, Andreas, Barghorn, Stefan, Lehmann, Alexander, Schoemaker, Hans, Hillen, Heinz, Gross, Gerhard, Ebert, Ulrich, Bruehl, Claus
Publicado no J Neurosci (2008)

Generation and Therapeutic Efficacy of Highly Oligomer-Specific β-Amyloid Antibodies

Por Hillen, Heinz, Barghorn, Stefan, Striebinger, Andreas, Labkovsky, Boris, Müller, Reinhold, Nimmrich, Volker, Nolte, Marc W., Perez-Cruz, Claudia, van der Auwera, Ingrid, van Leuven, Fred, van Gaalen, Marcel, Bespalov, Anton Y., Schoemaker, Hans, Sullivan, James P., Ebert, Ulrich
Publicado no J Neurosci (2010)