טוען...
Aβ Inhibition of Ionic Conductance in Mouse Basal Forebrain Neurons Is Dependent upon the Cellular Prion Protein PrP(C)
Current therapies for Alzheimer's disease (AD) address a loss of cholinergic neurons, while accumulation of neurotoxic amyloid β (Aβ) peptide assemblies is thought central to molecular pathogenesis. Overlaps may exist between prionopathies and AD wherein Aβ oligomers bind to the cellular prion...
שמור ב:
| הוצא לאור ב: | J Neurosci |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Society for Neuroscience
2011
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6633251/ https://ncbi.nlm.nih.gov/pubmed/22072680 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4367-11.2011 |
| תגים: |
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