Restoration of Synaptic Plasticity and Learning in Young and Aged NCAM-Deficient Mice by Enhancing Neurotransmission Mediated by GluN2A-Containing NMDA Receptors

Ítems similars

• Neural Cell Adhesion Molecule-Associated Polysialic Acid Regulates Synaptic Plasticity and Learning by Restraining the Signaling through GluN2B-Containing NMDA Receptors
  per: Kochlamazashvili, Gaga, et al.
  Publicat: (2010)
• Polysialylated Neural Cell Adhesion Molecule Is Involved in Induction of Long-Term Potentiation and Memory Acquisition and Consolidation in a Fear-Conditioning Paradigm
  per: Senkov, Oleg, et al.
  Publicat: (2006)
• Hippocampal Metaplasticity Induced by Deficiency in the Extracellular Matrix Glycoprotein Tenascin-R
  per: Bukalo, Olena, et al.
  Publicat: (2007)
• Transgenic mice overexpressing the extracellular domain of NCAM are impaired in working memory and cortical plasticity
  per: Brennaman, Leann H., et al.
  Publicat: (2011)
• The Extracellular Matrix Molecule Hyaluronic Acid Regulates Hippocampal Synaptic Plasticity by Modulating Postsynaptic L-Type Ca(2+) Channels
  per: Kochlamazashvili, Gaga, et al.
  Publicat: (2010)