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PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding, and Ca(2+) Signaling, Which Requires Ser332/334 in the CaM-Binding C Terminus
The Ca(2+)-dependent K(+) channel, KCa3.1 (KCNN4/IK/SK4), is widely expressed and contributes to cell functions that include volume regulation, migration, membrane potential, and excitability. KCa3.1 is now considered a therapeutic target for several diseases, including CNS disorders involving micro...
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| Publicado no: | J Neurosci |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Society for Neuroscience
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6608312/ https://ncbi.nlm.nih.gov/pubmed/25274816 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1008-14.2014 |
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