Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injury

Traumatic brain injury (TBI) has become a major health concern worldwide, and the poor outcome of TBI increases the need for therapeutic improvement. Secondary injuries following TBI, including excitotoxicity, lead to synaptic dysfunction and provide potential targets for intervention. Postsynaptic...

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Bibliografiske detaljer
Udgivet i:Cell Death Dis
Main Authors: Luo, Peng, Li, Xin, Wu, Xiuquan, Dai, Shuhui, Yang, Yuefan, Xu, Haoxiang, Jing, Da, Rao, Wei, Xu, Hongyu, Gao, Xiangyu, Fei, Zhou, Lu, Hongbing
Format: Artigo
Sprog:Inglês
Udgivet: Nature Publishing Group UK 2019
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Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6591282/
https://ncbi.nlm.nih.gov/pubmed/31235685
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1731-x
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