Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injury

Traumatic brain injury (TBI) has become a major health concern worldwide, and the poor outcome of TBI increases the need for therapeutic improvement. Secondary injuries following TBI, including excitotoxicity, lead to synaptic dysfunction and provide potential targets for intervention. Postsynaptic...

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Pubblicato in:Cell Death Dis
Autori principali: Luo, Peng, Li, Xin, Wu, Xiuquan, Dai, Shuhui, Yang, Yuefan, Xu, Haoxiang, Jing, Da, Rao, Wei, Xu, Hongyu, Gao, Xiangyu, Fei, Zhou, Lu, Hongbing
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group UK 2019
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Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6591282/
https://ncbi.nlm.nih.gov/pubmed/31235685
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1731-x
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