Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injury

Traumatic brain injury (TBI) has become a major health concern worldwide, and the poor outcome of TBI increases the need for therapeutic improvement. Secondary injuries following TBI, including excitotoxicity, lead to synaptic dysfunction and provide potential targets for intervention. Postsynaptic...

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Detalhes bibliográficos
Publicado no:Cell Death Dis
Main Authors: Luo, Peng, Li, Xin, Wu, Xiuquan, Dai, Shuhui, Yang, Yuefan, Xu, Haoxiang, Jing, Da, Rao, Wei, Xu, Hongyu, Gao, Xiangyu, Fei, Zhou, Lu, Hongbing
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6591282/
https://ncbi.nlm.nih.gov/pubmed/31235685
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1731-x
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