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“Dual Disease” TgAD/GSS mice exhibit enhanced Alzheimer’s disease pathology and reveal PrP(C)-dependent secretion of Aβ

To address the question of cross-talk between prion protein (PrP) and Alzheimer’s disease (AD), we generated TgAD/GSS mice that develop amyloid-β (Aβ) plaques of AD and PrP (specifically mutated PrP(A116V)) plaques of Gerstmann-Sträussler-Scheinker disease (GSS) and compared plaque-related features...

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Publicado en:Sci Rep
Autores principales: Qin, Kefeng, Zhao, Lili, Gregory, Crystal, Solanki, Ani, Mastrianni, James A.
Formato: Artigo
Lenguaje:Inglês
Publicado: Nature Publishing Group UK 2019
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6562043/
https://ncbi.nlm.nih.gov/pubmed/31189938
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-019-44317-w
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