Tsg101 positively regulates physiologic-like cardiac hypertrophy through FIP3-mediated endosomal recycling of IGF-1R

Development of physiologic cardiac hypertrophy has primarily been ascribed to the IGF-1 and its receptor, IGF-1 receptor (IGF-1R), and subsequent activation of the protein kinase B (Akt) pathway. However, regulation of endosome-mediated recycling and degradation of IGF-1R during physiologic hypertro...

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Detalhes bibliográficos
Publicado no:FASEB J
Main Authors: Essandoh, Kobina, Deng, Shan, Wang, Xiaohong, Jiang, Min, Mu, Xingjiang, Peng, Jiangtong, Li, Yutian, Peng, Tianqing, Wagner, Kay-Uwe, Rubinstein, Jack, Fan, Guo-Chang
Formato: Artigo
Idioma:Inglês
Publicado em: Federation of American Societies for Experimental Biology 2019
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6529337/
https://ncbi.nlm.nih.gov/pubmed/30884248
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201802338RR
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