A289 THE CROHN’S DISEASE-ASSOCIATED ADHERENT-INVASIVE E. COLI INDUCES MITOCHONDRIAL FRAGMENTATION IN ENTERIC EPITHELIUM THAT IS NOT DEPENDENT ON BACTERIAL SOLUBLE PRODUCTS OR MITOCHONDRIAL REACTIVE OXYGEN SPECIES

Lignende værker

• A155 THE IMMUNOMODULATOR, LEFLUNOMIDE, REDUCES MITOCHONDRIAL FRAGMENTATION CAUSED BY ADHERENT–INVASIVE E. COLI AND PERSISTENCE OF THE PATHOBIONT WITHIN ENTERIC EPITHELIA
  af: Mancini, N, et al.
  Udgivet: (2019)
• A273 THE CROHN’S DISEASE-ASSOCIATED PATHOBIONT ADHERENT-INVASIVE E. COLI (AIEC) INDUCES MITOCHONDRIAL FISSION IN EPITHELIAL CELLS IN ADVANCE OF APOPTOSIS
  af: Mancini, N, et al.
  Udgivet: (2018)
• Crohn’s Disease Pathobiont Adherent-Invasive E coli Disrupts Epithelial Mitochondrial Networks With Implications for Gut Permeability
  af: Mancini, Nicole L., et al.
  Udgivet: (2020)
• Indomethacin-induced translocation of bacteria across enteric epithelia is reactive oxygen species-dependent and reduced by vitamin C
  af: Schoultz, Ida, et al.
  Udgivet: (2012)
• Regulation of acid-base transporters by reactive oxygen species following mitochondrial fragmentation
  af: Johnson, David, et al.
  Udgivet: (2012)